MGZ Medizinisch Genetisches Zentrum

Hereditary Motor and Sensory Neuropathy, Overview, HMSN, CMT

Klinische Symptomatik

HMSN (hereditary motor and sensory neuropathy) or CMT (Charcot-Marie-Tooth) is a group of clinically and genetically heterogenous disorders of the peripheral nervous system. Patients develop symmetric, distally pronounced pareses and muscle atrophies, frequently accompanied by sensory impairment, reduced or absent deep tendon reflexes and foot deformities (e. g. pes cavus). This disease frequently results in an increasing handicap with age; in some cases, a wheelchair is required.
A prevalence of 1: 2 500 - 7: 100 000 indicates that these are the most frequent inherited neuromuscular disorders.

 

Clinical diagnosis

  • Positive family history; pattern of inheritance or sporadic
  • Characteristic symptoms (distal > proximal atrophy, gait disturbance, foot deformities, hypesthesia, reduced vibration sense, etc.)
  • Electrophysiological examination:

Nerve conduction velocity (NCV) > 38 m/s; reduced amplitudes: primary axonal neuropathy
Nerve conduction velocity (NCV) < 38 m/s: primary demyelinating neuropathy
Electromyogramm (EMG)

  • Sural nerve biopsy (if required)

 

Classification:
The demyelinating types, indicated by reduced nerve conduction velocity, are clinically differentiated from the axonal types, which are characterized by normal NCV and reduced amplitudes. Recent findings point to intermediate neuropathies that show signs of myelin- as well as axonal impairment. Further classifications take the predominant symptoms, family history, and causative mutations as well as the associated chromosomal loci into account. In later stages of the disease, axonal damage is common to almost all subtypes. The follwing table is based on the traditional classification according to Dyck and Lambert.

 

Type Synonyms Age of onset (years) Symptoms
HMSN1, CMT1

Hypertrophic Form of peroneal muscle atrophy
Charcot-Marie-Tooth
10 - 30
Demyelinating polyneuropathy, lslowly progredient distal pareses, frequently autosomal dominant
HMSN2, CMT2
Neuron form of peroneal muscle atrophie
Charcot-Marie-Tooth
20 - 40
Axonal neuropathie, clinical signs similiar to HMSN 1, frequently autosomal dominant
HMSN3, DSS
Hypertrophic neuropathy
Dejerine-Sottas-Syndrom
< 15
Demyelinating neuropathie,
progredient pareses,
pronounced sensory loss, trophic impairment,
frequently autosomal rezessiv

Congenital hypomyelinating neuropathy (CHN) is the most severe inherited form to date, manifest in newborns as well as in early childhood.
All Mendelian patterns of inheritance have been observed:

  • Autosomal dominant and recessive
  • X-chromosomal dominant and recessive


Multiple gene mutations and loci are known. Additional symptoms (deafness, pupillary anomalies, optic atrophy or visual loss, scoliosis, diaphragm impairment, vocal cord paresis, etc.) may point to the underlying gene and mutation. Particular mutations, however, may result in a broad spectrum of phenotypes, partially overlapping with other diseases - e. g., Friedreich Ataxia, Roussy-Levy syndrome, distal hereditary neuropathy or hereditary spastic paraparesis. Amyotrophic lateral sclerosis is also known as a spinal form of CMT. Severe courses of the disease are also designated as Dejerine-Sottas Syndrome (DSS) or Dejerine Sottas Neuropathy (DSN), as well as Congenital Hypomyelinating Neuropathy (CHN). A further subgroup comprises the hereditary sensory and autonomous neuropathies, most frequently disorders of early childhood with severe progression and additional symptoms (insensitivity to pain, anhidrosis, etc.). It is therefore not always possible to clinically define the underlying genetic defect. In such cases, differential genetic diagnostics may be of help.
The Medial Genetic Centre offers analyses covering nearly all known CMT genes:

Analyses for HMSN, CMT, DSN, CHN, HS(A)N at the MGZ

Type Inheritance Locus
Gene
HMSN type 1 - primaryly demyelinating
CMT1A (1E with deafness)
AD
17p12
PMP22
CMT1B, CHN, DSS, intermediate form
AD
1q22.31
MPZ
CMTX, intermediate form XR/XD Xq13.1 GJB1/Cx32
CMT1D, CHN, DSS AD 10q21.3 EGR2
CMT1F AD 8p21 NEFL
CMT1C AD 16p13.13 LITAF/SIMPLE
CMT4A AR 8q21.11 GDAP1
CMT4C AR 5q32 SH3TC2
CMT4D (HMSNL) AR 8q24.3 NDRG1
CMT4F (DSS) AR 19q13.2 PRX
CCFDN AR 18q23 CTDP1
HMSN type II, primaryly axonal
CMT2A2 AD 1p36.2 MFN2
CMT2I, J (deafness, pupillary anomalies) AD 1q23.3 MPZ
CMTX, intermediate form XR/XD Xq13.1 GJB1/Cx32
CMT2B (ulcerations) AD 3q21.3 RAB7
CMT2D, dHMN 5 AD 7p15 GARS
CMT2E AD 8p21 NEFL
CMT2F AD 7q11.23 HSPB1
CMT2K AD/AR 8q21.11 GDAP1
CMT2L AD 12q24 HSPB8
CMT2B1 AR 1q22 LMNA
HMSN type III, juvenile form of HMSN I
DSS/CHN AD/AR 17p12 PMP22
DSS/CHN AD/AR 1q23.3 MPZ
DSS/CHN AD/AR 10q21.3 EGR2
DSS AR 19q13.2 PRX
HMSN intermediate forms
DI-CMTB AD 19p13.2 DNM2
DI-CMTD AD 1q23.3 MPZ
CMTX XR/XD Xq13.1 GJB1/Cx32
CMTRIA AR 8q21.1 GDAP1
Distal hereditary motor europathies HMN
Distal HMN 2, CMT2L AD 12q24 HSPB8
Distal HMN 5, CMT2D AD 7p15 GARS
Distal HMN 7B/ALS associated AD 2p13 DCTN1
Distal HMN/ALS4 AD 9q34 SETX
Distal HMN/Silver Syndrome/SPG17/DSMAV AD 11q13 BSCL2
Hereditary recurrend focal neuropathies
HNPP
AD
17p12
PMP22
HNA AD 17q25 SEPT9
Giant Axon Neuropathie, GAN1 AR 16q24.1 GAN
Hereditary sensory (autonomous) neuropathies
HSAN 1 AD 9p22.31 SPTLC1
HSAN 4 (CIPA) AR 1q23.1 NTRK1
HSAN 5 AR 1p13.1 NGFB

 

AR autosomal recessive
AD autosomal dominant
XD X-linked dominant
XR X-linked recessive
DI-CMT dominant-intermediate Charcot-Marie-Tooth-disease
CCFDN Congenital Cataracts, Facial Dysmorphism & Neuropathy Syndrome
HNPP Hereditary Neuropathy with Liability to Pressure Palsies
HNA Hereditary neuralgic Amyotrophy
CHN Congenital Hypomyelination Neuropathy
DSS Dejerine-Sottas-Syndrome
CIPA Congenital Insensitivity to Pain with Anhidrosis

 

 





MGZ
Medizinisch Genetisches Zentrum

Bayerstraße 3-5 (durch die Mathäser-Passage)

Eingang Schlosserstraße 6
80335 München
info@mgz-muenchen.de
Tel. +49 (0)89/30 90 886-0
Fax +49 (0)89/30 90 886-66

Ärztliche Leitung
Prof. Dr. med. Dipl. chem. Elke Holinski-Feder